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Erratum: The evolutionary turnover of recombination hot spots contributes to speciation in mice.

Genes & development

Smagulova F, Brick K, Pu Y, Camerini-Otero RD, Petukhova GV.
PMID: 27036969
Genes Dev. 2016 Apr 01;30(7):871. doi: 10.1101/gad.280677.116.

Due to a technical error in processing the figures in the above-mentioned article, Figures 3, A and B; 4B; 5B; and 6, A and C contained errors or missing elements. The errors have been corrected in both the PDF...

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Smagulova F, Brick K, Pu Y, et al. Erratum: The evolutionary turnover of recombination hot spots contributes to speciation in mice. Genes Dev. 2016;30(7):871doi: 10.1101/gad.280677.116.
Smagulova, F., Brick, K., Pu, Y., Camerini-Otero, R. D., & Petukhova, G. V. (2016). Erratum: The evolutionary turnover of recombination hot spots contributes to speciation in mice. Genes & development, 30(7), 871. https://doi.org/10.1101/gad.280677.116
Smagulova, Fatima, et al. "Erratum: The evolutionary turnover of recombination hot spots contributes to speciation in mice." Genes & development vol. 30,7 (2016): 871. doi: https://doi.org/10.1101/gad.280677.116
Smagulova F, Brick K, Pu Y, Camerini-Otero RD, Petukhova GV. Erratum: The evolutionary turnover of recombination hot spots contributes to speciation in mice. Genes Dev. 2016 Apr 01;30(7):871. doi: 10.1101/gad.280677.116. PMID: 27036969; PMCID: PMC4826402.
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Corrigendum: inhibition of α-KG-dependent histone and DNA demethylases by fumarate and succinate that are accumulated in mutations of FH and SDH tumor suppressors.

Genes & development

Xiao M, Yang H, Xu W, Ma S, Lin H, Zhu H, Liu L, Liu Y, Yang C, Xu Y, Zhao S, Ye D, Xiong Y, Guan KL.
PMID: 25877923
Genes Dev. 2015 Apr 15;29(8):887. Epub 2015 Apr 15.

No abstract available.

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Xiao M, Yang H, Xu W, et al. Corrigendum: inhibition of α-KG-dependent histone and DNA demethylases by fumarate and succinate that are accumulated in mutations of FH and SDH tumor suppressors. Genes Dev. 2015;29(8):887
Xiao, M., Yang, H., Xu, W., Ma, S., Lin, H., Zhu, H., Liu, L., Liu, Y., Yang, C., Xu, Y., Zhao, S., Ye, D., Xiong, Y., & Guan, K. L. (2015). Corrigendum: inhibition of α-KG-dependent histone and DNA demethylases by fumarate and succinate that are accumulated in mutations of FH and SDH tumor suppressors. Genes & development, 29(8), 887.
Xiao, Mengtao, et al. "Corrigendum: inhibition of α-KG-dependent histone and DNA demethylases by fumarate and succinate that are accumulated in mutations of FH and SDH tumor suppressors." Genes & development vol. 29,8 (2015): 887.
Xiao M, Yang H, Xu W, Ma S, Lin H, Zhu H, Liu L, Liu Y, Yang C, Xu Y, Zhao S, Ye D, Xiong Y, Guan KL. Corrigendum: inhibition of α-KG-dependent histone and DNA demethylases by fumarate and succinate that are accumulated in mutations of FH and SDH tumor suppressors. Genes Dev. 2015 Apr 15;29(8):887. Epub 2015 Apr 15. PMID: 25877923; PMCID: PMC4403263.
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Modularity: genes, development and evolution.

Annual review of ecology, evolution, and systematics

Melo D, Porto A, Cheverud JM, Marroig G.
PMID: 28966564
Annu Rev Ecol Evol Syst. 2016;47:463-486. doi: 10.1146/annurev-ecolsys-121415-032409. Epub 2016 Sep 07.

Modularity has emerged as a central concept for evolutionary biology, providing the field with a theory of organismal structure and variation. This theory has reframed long standing questions and serves as a unified conceptual framework for genetics, developmental biology...

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Melo D, Porto A, Cheverud JM, et al. Modularity: genes, development and evolution. Annu Rev Ecol Evol Syst. 2016;47:463-486doi: 10.1146/annurev-ecolsys-121415-032409.
Melo, D., Porto, A., Cheverud, J. M., & Marroig, G. (2016). Modularity: genes, development and evolution. Annual review of ecology, evolution, and systematics, 47463-486. https://doi.org/10.1146/annurev-ecolsys-121415-032409
Melo, Diogo, et al. "Modularity: genes, development and evolution." Annual review of ecology, evolution, and systematics vol. 47 (2016): 463-486. doi: https://doi.org/10.1146/annurev-ecolsys-121415-032409
Melo D, Porto A, Cheverud JM, Marroig G. Modularity: genes, development and evolution. Annu Rev Ecol Evol Syst. 2016;47:463-486. doi: 10.1146/annurev-ecolsys-121415-032409. Epub 2016 Sep 07. PMID: 28966564; PMCID: PMC5617135.
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Spliceosome activation: U4 is the path, stem I is the goal, and Prp8 is the keeper. Let's cheer for the ATPase Brr2!.

Genes & development

Nielsen KH, Staley JP.
PMID: 23154979
Genes Dev. 2012 Nov 15;26(22):2461-7. doi: 10.1101/gad.207514.112.

During pre-mRNA splicing, the spliceosome is activated for catalysis by unwinding base-paired U4/U6 small nuclear RNAs, a step that must be precisely timed. We know that unwinding requires the ATPase Brr2, but the mechanism and regulation of unwinding have...

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Nielsen KH, Staley JP. Spliceosome activation: U4 is the path, stem I is the goal, and Prp8 is the keeper. Let's cheer for the ATPase Brr2!. Genes Dev. 2012;26(22):2461-7doi: 10.1101/gad.207514.112.
Nielsen, K. H., & Staley, J. P. (2012). Spliceosome activation: U4 is the path, stem I is the goal, and Prp8 is the keeper. Let's cheer for the ATPase Brr2!. Genes & development, 26(22), 2461-7. https://doi.org/10.1101/gad.207514.112
Nielsen, Klaus H, and Staley, Jonathan P. "Spliceosome activation: U4 is the path, stem I is the goal, and Prp8 is the keeper. Let's cheer for the ATPase Brr2!." Genes & development vol. 26,22 (2012): 2461-7. doi: https://doi.org/10.1101/gad.207514.112
Nielsen KH, Staley JP. Spliceosome activation: U4 is the path, stem I is the goal, and Prp8 is the keeper. Let's cheer for the ATPase Brr2!. Genes Dev. 2012 Nov 15;26(22):2461-7. doi: 10.1101/gad.207514.112. PMID: 23154979; PMCID: PMC3505815.
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Quit your YAPing: a new target for cancer therapy.

Genes & development

Stanger BZ.
PMID: 22713867
Genes Dev. 2012 Jun 15;26(12):1263-7. doi: 10.1101/gad.196501.112.

The Hippo pathway is an evolutionarily conserved signaling module that plays multiple roles in embryonic development. Components of the pathway, which includes a kinase cascade and a downstream complex composed of YAP and TEAD transcription factors, are dysregulated in...

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Stanger BZ. Quit your YAPing: a new target for cancer therapy. Genes Dev. 2012;26(12):1263-7doi: 10.1101/gad.196501.112.
Stanger, B. Z. (2012). Quit your YAPing: a new target for cancer therapy. Genes & development, 26(12), 1263-7. https://doi.org/10.1101/gad.196501.112
Stanger, Ben Z. "Quit your YAPing: a new target for cancer therapy." Genes & development vol. 26,12 (2012): 1263-7. doi: https://doi.org/10.1101/gad.196501.112
Stanger BZ. Quit your YAPing: a new target for cancer therapy. Genes Dev. 2012 Jun 15;26(12):1263-7. doi: 10.1101/gad.196501.112. PMID: 22713867; PMCID: PMC3387654.
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Corrigendum: Transcription elongation rate affects nascent histone pre-mRNA folding and 3' end processing.

Genes & development

Saldi T, Fong N, Bentley DL.
PMID: 29692357
Genes Dev. 2018 Apr 01;32(7):592. doi: 10.1101/gad.314948.118.

No abstract available.

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Saldi T, Fong N, Bentley DL. Corrigendum: Transcription elongation rate affects nascent histone pre-mRNA folding and 3' end processing. Genes Dev. 2018;32(7):592doi: 10.1101/gad.314948.118.
Saldi, T., Fong, N., & Bentley, D. L. (2018). Corrigendum: Transcription elongation rate affects nascent histone pre-mRNA folding and 3' end processing. Genes & development, 32(7), 592. https://doi.org/10.1101/gad.314948.118
Saldi, Tassa, et al. "Corrigendum: Transcription elongation rate affects nascent histone pre-mRNA folding and 3' end processing." Genes & development vol. 32,7 (2018): 592. doi: https://doi.org/10.1101/gad.314948.118
Saldi T, Fong N, Bentley DL. Corrigendum: Transcription elongation rate affects nascent histone pre-mRNA folding and 3' end processing. Genes Dev. 2018 Apr 01;32(7):592. doi: 10.1101/gad.314948.118. PMID: 29692357; PMCID: PMC5959241.
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Corrigendum: Coupled enhancer and coding sequence evolution of a homeobox gene shaped leaf diversity.

Genes & development

Vuolo F, Mentink RA, Hajheidari M, Bailey CD, Filatov DA, Tsiantis M.
PMID: 29237742
Genes Dev. 2017 Nov 01;31(21):2199. doi: 10.1101/gad.309179.117.

No abstract available.

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Vuolo F, Mentink RA, Hajheidari M, et al. Corrigendum: Coupled enhancer and coding sequence evolution of a homeobox gene shaped leaf diversity. Genes Dev. 2017;31(21):2199doi: 10.1101/gad.309179.117.
Vuolo, F., Mentink, R. A., Hajheidari, M., Bailey, C. D., Filatov, D. A., & Tsiantis, M. (2017). Corrigendum: Coupled enhancer and coding sequence evolution of a homeobox gene shaped leaf diversity. Genes & development, 31(21), 2199. https://doi.org/10.1101/gad.309179.117
Vuolo, Francesco, et al. "Corrigendum: Coupled enhancer and coding sequence evolution of a homeobox gene shaped leaf diversity." Genes & development vol. 31,21 (2017): 2199. doi: https://doi.org/10.1101/gad.309179.117
Vuolo F, Mentink RA, Hajheidari M, Bailey CD, Filatov DA, Tsiantis M. Corrigendum: Coupled enhancer and coding sequence evolution of a homeobox gene shaped leaf diversity. Genes Dev. 2017 Nov 01;31(21):2199. doi: 10.1101/gad.309179.117. PMID: 29237742; PMCID: PMC5749167.
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Corrigendum: In vivo investigation of the transcription, processing, endonucleolytic activity, and functional relevance of the spatial distribution of a plant miRNA.

Genes & development

Parizotto EA, Dunoyer P, Rahm N, Himber C, Voinnet O.
PMID: 27222518
Genes Dev. 2016 May 15;30(10):1251-2. doi: 10.1101/gad.283721.116.

No abstract available.

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Parizotto EA, Dunoyer P, Rahm N, et al. Corrigendum: In vivo investigation of the transcription, processing, endonucleolytic activity, and functional relevance of the spatial distribution of a plant miRNA. Genes Dev. 2016;30(10):1251-2doi: 10.1101/gad.283721.116.
Parizotto, E. A., Dunoyer, P., Rahm, N., Himber, C., & Voinnet, O. (2016). Corrigendum: In vivo investigation of the transcription, processing, endonucleolytic activity, and functional relevance of the spatial distribution of a plant miRNA. Genes & development, 30(10), 1251-2. https://doi.org/10.1101/gad.283721.116
Parizotto, Eneida Abreu, et al. "Corrigendum: In vivo investigation of the transcription, processing, endonucleolytic activity, and functional relevance of the spatial distribution of a plant miRNA." Genes & development vol. 30,10 (2016): 1251-2. doi: https://doi.org/10.1101/gad.283721.116
Parizotto EA, Dunoyer P, Rahm N, Himber C, Voinnet O. Corrigendum: In vivo investigation of the transcription, processing, endonucleolytic activity, and functional relevance of the spatial distribution of a plant miRNA. Genes Dev. 2016 May 15;30(10):1251-2. doi: 10.1101/gad.283721.116. PMID: 27222518; PMCID: PMC4888844.
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Erratum: Set1/COMPASS repels heterochromatin invasion at euchromatic sites by disrupting Suv39/Clr4 activity and nucleosome stability.

Genes & development

Greenstein RA, Barrales RR, Sanchez NA, Bisanz JE, Braun S, Al-Sady B.
PMID: 32747479
Genes Dev. 2020 Aug 01;34(15):1106. doi: 10.1101/gad.342006.120.

No abstract available.

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Greenstein RA, Barrales RR, Sanchez NA, et al. Erratum: Set1/COMPASS repels heterochromatin invasion at euchromatic sites by disrupting Suv39/Clr4 activity and nucleosome stability. Genes Dev. 2020;34(15):1106doi: 10.1101/gad.342006.120.
Greenstein, R. A., Barrales, R. R., Sanchez, N. A., Bisanz, J. E., Braun, S., & Al-Sady, B. (2020). Erratum: Set1/COMPASS repels heterochromatin invasion at euchromatic sites by disrupting Suv39/Clr4 activity and nucleosome stability. Genes & development, 34(15), 1106. https://doi.org/10.1101/gad.342006.120
Greenstein, R A, et al. "Erratum: Set1/COMPASS repels heterochromatin invasion at euchromatic sites by disrupting Suv39/Clr4 activity and nucleosome stability." Genes & development vol. 34,15 (2020): 1106. doi: https://doi.org/10.1101/gad.342006.120
Greenstein RA, Barrales RR, Sanchez NA, Bisanz JE, Braun S, Al-Sady B. Erratum: Set1/COMPASS repels heterochromatin invasion at euchromatic sites by disrupting Suv39/Clr4 activity and nucleosome stability. Genes Dev. 2020 Aug 01;34(15):1106. doi: 10.1101/gad.342006.120. PMID: 32747479; PMCID: PMC7397851.
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Hierarchical reactivation of transcription during mitosis-to-G1 transition by Brn2 and Ascl1 in neural stem cells.

Genes & development

Soares MAF, Soares DS, Teixeira V, Heskol A, Bressan RB, Pollard SM, Oliveira RA, Castro DS.
PMID: 34168041
Genes Dev. 2021 Jul 01;35(13):1020-1034. doi: 10.1101/gad.348174.120. Epub 2021 Jun 24.

During mitosis, chromatin condensation is accompanied by a global arrest of transcription. Recent studies suggest transcriptional reactivation upon mitotic exit occurs in temporally coordinated waves, but the underlying regulatory principles have yet to be elucidated. In particular, the contribution...

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Soares MAF, Soares DS, Teixeira V, et al. Hierarchical reactivation of transcription during mitosis-to-G1 transition by Brn2 and Ascl1 in neural stem cells. Genes Dev. 2021;35(13):1020-1034doi: 10.1101/gad.348174.120.
Soares, M. A. F., Soares, D. S., Teixeira, V., Heskol, A., Bressan, R. B., Pollard, S. M., Oliveira, R. A., & Castro, D. S. (2021). Hierarchical reactivation of transcription during mitosis-to-G1 transition by Brn2 and Ascl1 in neural stem cells. Genes & development, 35(13), 1020-1034. https://doi.org/10.1101/gad.348174.120
Soares, Mário A F, et al. "Hierarchical reactivation of transcription during mitosis-to-G1 transition by Brn2 and Ascl1 in neural stem cells." Genes & development vol. 35,13 (2021): 1020-1034. doi: https://doi.org/10.1101/gad.348174.120
Soares MAF, Soares DS, Teixeira V, Heskol A, Bressan RB, Pollard SM, Oliveira RA, Castro DS. Hierarchical reactivation of transcription during mitosis-to-G1 transition by Brn2 and Ascl1 in neural stem cells. Genes Dev. 2021 Jul 01;35(13):1020-1034. doi: 10.1101/gad.348174.120. Epub 2021 Jun 24. PMID: 34168041; PMCID: PMC8247608.
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Time is of the essence: the molecular mechanisms of primary microcephaly.

Genes & development

Phan TP, Holland AJ.
PMID: 34862179
Genes Dev. 2021 Dec 01;35(23):1551-1578. doi: 10.1101/gad.348866.121.

Primary microcephaly is a brain growth disorder characterized by a severe reduction of brain size and thinning of the cerebral cortex. Many primary microcephaly mutations occur in genes that encode centrosome proteins, highlighting an important role for centrosomes in...

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Phan TP, Holland AJ. Time is of the essence: the molecular mechanisms of primary microcephaly. Genes Dev. 2021;35(23):1551-1578doi: 10.1101/gad.348866.121.
Phan, T. P., & Holland, A. J. (2021). Time is of the essence: the molecular mechanisms of primary microcephaly. Genes & development, 35(23), 1551-1578. https://doi.org/10.1101/gad.348866.121
Phan, Thao P, and Holland, Andrew J. "Time is of the essence: the molecular mechanisms of primary microcephaly." Genes & development vol. 35,23 (2021): 1551-1578. doi: https://doi.org/10.1101/gad.348866.121
Phan TP, Holland AJ. Time is of the essence: the molecular mechanisms of primary microcephaly. Genes Dev. 2021 Dec 01;35(23):1551-1578. doi: 10.1101/gad.348866.121. PMID: 34862179.
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The evolution of metazoan shelterin.

Genes & development

Myler LR, Kinzig CG, Sasi NK, Zakusilo G, Cai SW, de Lange T.
PMID: 34764137
Genes Dev. 2021 Dec 01;35(23):1625-1641. doi: 10.1101/gad.348835.121. Epub 2021 Nov 11.

The mammalian telomeric shelterin complex-comprised of TRF1, TRF2, Rap1, TIN2, TPP1, and POT1-blocks the DNA damage response at chromosome ends and interacts with telomerase and the CST complex to regulate telomere length. The evolutionary origins of shelterin are unclear,...

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Myler LR, Kinzig CG, Sasi NK, et al. The evolution of metazoan shelterin. Genes Dev. 2021;35(23):1625-1641doi: 10.1101/gad.348835.121.
Myler, L. R., Kinzig, C. G., Sasi, N. K., Zakusilo, G., Cai, S. W., & de Lange, T. (2021). The evolution of metazoan shelterin. Genes & development, 35(23), 1625-1641. https://doi.org/10.1101/gad.348835.121
Myler, Logan R, et al. "The evolution of metazoan shelterin." Genes & development vol. 35,23 (2021): 1625-1641. doi: https://doi.org/10.1101/gad.348835.121
Myler LR, Kinzig CG, Sasi NK, Zakusilo G, Cai SW, de Lange T. The evolution of metazoan shelterin. Genes Dev. 2021 Dec 01;35(23):1625-1641. doi: 10.1101/gad.348835.121. Epub 2021 Nov 11. PMID: 34764137.
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Showing 1 to 12 of 219 entries